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AG1 is not a relA1 mutant. In (A) and (B), relA over-expressing cells of the ASKA library accumulate WT glycogen content. In (C) and (D), relA expression vector of the ASKA library complements the glycogen-deficient phenotype of ΔrelA cells of the Keio collection. ΔglgA cells of the Keio collection are used as negative control for glycogen accumulation.
