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. 2010 Feb 3;298(4):R862–R869. doi: 10.1152/ajpregu.00575.2009

Fig. 5.

Fig. 5.

A: quantitation of EETs in the perfusate collected from shear stress (2 and 10 dyn/cm2)-stimulated mesenteric arteries of female eNOS-KO mice in control conditions (n/n = 5/9), in the presence of 6-(2-proparglyoxyphenyl) hexanoic acid (PPOH; n/n = 5/7), and transfected with CYP2C29 siRNA (n/n = 5/12), or nonsilencing siRNA (n/n = 5/7) for 72 h, respectively. *P < 0.05 compared with control. †P < 0.05 compared to nonsilencing siRNA. B: shear stress (10 dyn/cm2)-induced vasodilation in control conditions (n/n = 5/10) (day 1) and after transfection with CYP2C29 siRNA (n/n = 5/10) or nonsilencing siRNA (n/n = 5/10) for 72 h (day 4). *P < 0.05 compared with nonsilencing siRNA. C and D: CYP2C29 mRNA (C) and protein (D) expressions in mesenteric arteries in control conditions (including untransfected for zero and 72 h), and transfected with CYP 2C29 siRNA, or nonsilencing siRNA, at different time points.