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Changes within BAD pathway may elucidate a potential mechanism by which PAK6 mediates resistance to radiation-induced apoptosis. (a) Western blot analysis showing phosphorylation sites Ser112 and 136 on BAD as well as total BAD levels in both control and PAK6- knockdown PC3 cells. (b) Enhanced cytochrome c release in PAK6- knockdown PC3 cells compared to control cells 72 hours following 6 Gy irradiation.
