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. Author manuscript; available in PMC: 2011 Jun 1.

Published in final edited form as: J Mol Cell Cardiol. 2009 Oct 20;48(6):1071–1079. doi: 10.1016/j.yjmcc.2009.10.008

Fig. 6 — Absolute retention of ^99mTc-NC100692 (%ID/g) within myocardium was determined by GWC.

(A) ^99mTc-NC100692 retention in IGF-1 (n=5) and lacZ-control (n=3) rats at 4 weeks post-MI. There was a significant increase in retention of ^99mTc-NC100692 within infarct territory of all animals compared to remote segments. This increase in ^99mTc-NC100692 was significantly higher in IGF-1 animals than in lacZ-controls. This trend was true for all myocardial territories.

(B) ^99mTc-NC100692 retention at 16 weeks post-MI was significantly reduced in IGF-1 animals (n=5) compared to lacZ rats (n=6). The trend for increased ^99mTc-NC100692 retention in IGF-1 treated rats was also observed in border and remote regions.

*P<0.05 vs. lacZ, †P<0.05 vs. remote

Fig. 6 — Absolute retention of ^99mTc-NC100692 (%ID/g) within myocardium was determined by GWC.

(A) ^99mTc-NC100692 retention in IGF-1 (n=5) and lacZ-control (n=3) rats at 4 weeks post-MI. There was a significant increase in retention of ^99mTc-NC100692 within infarct territory of all animals compared to remote segments. This increase in ^99mTc-NC100692 was significantly higher in IGF-1 animals than in lacZ-controls. This trend was true for all myocardial territories.

(B) ^99mTc-NC100692 retention at 16 weeks post-MI was significantly reduced in IGF-1 animals (n=5) compared to lacZ rats (n=6). The trend for increased ^99mTc-NC100692 retention in IGF-1 treated rats was also observed in border and remote regions.

*P<0.05 vs. lacZ, †P<0.05 vs. remote