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. 2010 Jan 13;107(Suppl 1):1765–1771. doi: 10.1073/pnas.0906222107

Fig. 3.

Fig. 3.

Duplication of selected LCRs during molecular evolution of the primates (updated from ref. 122). The figure is not to scale. LCRJ, Opsin and TEX28 array at Xq28; LCR15, LCR highly repeated in chromosome 15q11-q14; LCRK, LCR flanking the genes FLNA and EMD at Xq28; PWS/AS, Prader–Willi and Angelman syndromes; DGS, DiGeorge syndrome; SMS, Smith–Magenis syndrome; WBS, Williams–Beuren syndrome; GBA, glucocerebrosidase gene; NEMO, gene mutated in incontinentia pigmenti; PMCHL1/2, chimeric genes derived from the melanin-concentrating hormone gene; NF1, neurofibromatosis 1; CMT1A, Charcot-Marie-Tooth disease type 1A; LCR16a, low copy repeats on chromosome 16; SMN2, gene mutated in spinal muscular atrophy. This figure was adapted, with permission, from ref. 122.