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. Author manuscript; available in PMC: 2010 Jun 1.
Published in final edited form as: J Hypertens. 2010 May;28(5):1034–1043. doi: 10.1097/hjh.0b013e328336ade3

Fig. 3.

Fig. 3

Representative immunohistochemistry images (a) and the antibody-positive area (b) for desmin, and glomerular expression of podocin (c) and nephrin (d) mRNA. Podocyte injury revealed by desmin and mRNA expression of glomerular podocin and nephrin was increased in SHR/ND, which was significantly improved by cilnidipine but not by amlodipine. *P < 0.05, **P < 0.01 vs. WKY, #P < 0.05 vs. SHR/ND, †P < 0.05 vs. SHR/ND + amlodipine (n = 11–12). SHR, spontaneously hypertensive rat; WKY, Wistar–Kyoto rats.