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. 2010 Jun;185(2):537–547. doi: 10.1534/genetics.109.113373

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Figure 2.— — Genomic assay results of the EGL-15 CTD role in SM chemoattraction. EGL-15 Y1009 and Y1087 are functionally redundant. Sex myoblast distributions for three lines for each construct are shown. The indicated transgenes were all tested in an egl-15(null) background. (A) Wild-type EGL-15 (NH#112). (B) A truncated version of EGL-15 that correlates to the egl-15(n1457) mutant (NH#838). (C) The single Y884F mutation fails to disrupt sex myoblast migration (NH#1379). (D and E) The single Y1009F (NH#818) or Y1087F (NH#1382) mutations do not account for the posterior SM positions of ΔCTD. (F and G) Double mutations with Y884 (Y884F/Y1009F, NH#841; Y884F/Y1087F, NH#1380) are not more affected than the respective single mutations. (H and I) The Y1009F mutation in conjunction with either the Y1087F mutation (NH#1370) or with both Y1087F and Y884F (NH#1356) (ΔSEM-5) abolishes SM chemoattraction. n, number of sex myoblasts scored.