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. Author manuscript; available in PMC: 2011 Aug 1.
Published in final edited form as: J Mol Cell Cardiol. 2010 Apr 1;49(2):322–329. doi: 10.1016/j.yjmcc.2010.03.017

Fig. 2.

Fig. 2

Effect of acute ethanol (EtOH) exposure on cell shortening and relengthening mechanics in cardiomyocytes from wild-type (WT) and ALDH2 knockout (ALDH-KO) mice. (A): Resting cell length; (B): Peak shortening amplitude (as a percentage of resting cell length); (C): Maximal velocity of shortening (+ dL/dt); (D): Maximal velocity of relengthening (−dL/dt); (E): Time-to- peak shortening (TPS); and (F): Time-to-90% relengthening (TR90). Mean ± SEM, n = 132–133 myocytes per group, *p < 0.05 vs. WT group, # p < 0.05 vs. WT+EtOH group.