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. 2010 Apr 2;298(6):H1671–H1678. doi: 10.1152/ajpheart.01028.2009

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Fig. 1. — Effect of chlorocyclopentyl adenosine (CCPA) and anti-Gβγ peptide on inositol 1,4,5-triphosphate (IP₃) produced in homogenates of hearts obtained from wild-type and adenosine A₁ receptor (A₁R) knockout (KO) mice. CCPA and peptide were administered at 1 and 50 μM, respectively. The A₁R antagonist 8-cyclopentyl-1,3-dipropylxanthine (DPCPX) was administered at 100 nM. Values are means ± SE for 3–4 hearts. *Mean is significantly different from the agent-free value (P < 0.05). †Means are significantly different from the CCPA-alone value (P < 0.05).