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. Author manuscript; available in PMC: 2010 Nov 1.
Published in final edited form as: Cell Metab. 2009 Nov;10(5):379–391. doi: 10.1016/j.cmet.2009.10.003

Table 1.

Genes differentially regulated by glucose treatment

A. Genes upregulated by glucose treatment
Transcript/Gene Brief description DAF-16 binding sites# (bp) GTAAAC/TA GATA transcription factor binding site (bp) CTTATCA
K09H11.7 Haloacid dehalogenase-like hydrolase −4863
dC53A3.2 Haloacid dehalogenase-like hydrolase −4908, −4873, −4189 −4550, −1559
C18H9.6 Unknown −4875 −430
F44E7.2 Haloacid dehalogenase-like hydrolase −2485, −1801, −1766 −2124, −636
K02G10.7 (aqp-8)* Major intrinsic protein (MIP), aquaporin −2919, −2080 −230
Y40B10A.6 o-methyltransferase domain 2021, −390
F21C10.9 Unknown −2937, −1952, −1637, −373
K11D12.4 Choline or carnitine o-acyltransferase −3785, −826, −380 −4348, −2735
dF21F8.4 Putative aspartyl (acid) protease −3035 4371, −4035, −3162, −2287, −1531, −1484
C17C3.1d Lipid storage −4547, −2753, −2033
Y69A2AR.4 (smf-3)* Solute carrier family 11 −3187, −207, −194
F10D2.11 UDP-glucoronosyl/glucosyl transferase −843, −366 4076, −3374, −2136, −12
Y53F4B.6 Growth, size, and locomotor behavior
Y40B10A.2 o-methyltransferase domain −2335 −1378
F15H10.1 (col-12) Collagen −4123, 3192, −1405 −3147
C29E6.5 (nhr-43)* Nuclear hormone receptor −4528, −3392 −1002, −943, −231
F31F4.5 Pseudogene
F09F3.9 Choline or carnitine o-acyltransferase −1395, −967, −42 −988
Y80D3A.7 (ptr-22) Patched superfamily −4270, −2815 −3331
F44G3.2 Plant ATP-guanido phosphotransferase −2651, −1490 −4345
F15B9.1 (far-3) Fatty acid- and retinol-binding protein 3 −1712, −421, −409 −4749, −979
C54D10.1 (cdr-2)* Cadmium-responsive gene 2 −4690, −2245, −2095, −2038, −1540, −1024
dC05E11.4 (amt-1) Ammonium transporter family −4905, −3889, −3671, −3440, −3246, −2591, −1606, 1327, −1240, −1118 −1789, −1461
B. Genes downregulated by glucose treatment
Transcript/Gene Brief description DAF-16 binding sites# (bp) GTAAAC/TA GATA transcription factor binding site (bp) CTTATCA
dZK6.7a Lipid storage, chromosome segregation, pathogen response −2695, −859 −4312, −3979, −80
C14C6.5 ShTK and CC domains
C50F7.5 Unknown −3658, −135 −1107, −1084, −240, −209
T20B12.4 Unknown
dF48D6.4a Unknown −4869, −4476, −4316, −1427 −3835, −403
dF32A5.5b (aqp-1)* Aquaporin −172 −2364, −2285, −112
dF48D6.4c Unknown −4869, −4476, −4316, −1427 −3835, −403
dZK6.7b Lipid storage, chromosome segregation, pathogen response −2695, −859 −4312, −3979, −80
dY4C6B.6 Glucosyl-ceramidases −3890, −3363, −2443, −1909 −155, −70
dF48D6.4b Unknown −4869, −4476, −4316, −1427 −3835, −403
dF15E11.14 Unknown −2707 −394
dF27C8.4 Downstream target of a VHL-1 pathway −4923, −2524, −992 −2810, −1883, −1007, −75
Y37A1B.12 (tor-1) May bind ATP −113, −59 −4039
T08A9.9 Similar to bactericidal amoebapores −4817, −3857, −348 −1039, −132
F59B2.11 Unknown −4056
C25B8.3a (cpr-6)* Cysteine protease domain −4500, −2119 −1972
C23H5.8a* Unknown −4036 −72

See Figure S8 for fold changes of these genes and Table S3 for fold changes of all the genes in microarray analysis.

d

Genes that were up- (or down-) regulated both by glucose treatment and by inactivation of daf-16.

*

The change in mRNA expression level upon glucose treatment was confirmed by qRT-PCR (Figure S9). The lifespan of deletion mutants for these genes was measured (Figure 4A and Figure S10)

#

We found that 37 of the 40 glucose-responsive transcripts (93%) contain one or more DAF-16 binding elements in their 5-kb upstream regulatory regions. This enrichment is statistically significant (P<0.05, hypergeometric probability) compared to that (78%) of random C. elegans genes (Kenyon and Murphy, 2006).