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. 2010 Apr 28;14(2):201. doi: 10.1186/cc8153

Table 3.

Etiology and investigation of post-cardiopulmonary bypass ventricular dysfunction

Cause Investigation Finding
General
 Exacerbation of preoperative ventricular dysfunction with relative intolerance to cardioplegic asystolic, hypoxic arrest TOE Global or regional wall motion abnormality
 Reperfusion injury TOE Global wall motion abnormality
 Inadequate myocardial protection (underlying coronary anatomy, route of cardioplegia, type of cardioplegia) TOE Global wall motion abnormality
Case/patient specific
 Ischaemia/infarction
  Vessel spasm (native coronaries, internal mammary artery) ECG, TOE, graft flow ECG changes, regional wall motion abnormality, poor graft flow
  Emboli (air, clot, particulate matter) ECG, TOE, graft flow ECG changes, regional wall motion abnormality, poor graft flow
  Technical graft anastomotic tissues ECG, TOE, graft flow ECG changes, regional wall motion abnormality, poor graft flow
  Kink/clotting of bypass grafts, native vessels ECG, TOE, graft flow, inspection ECG changes, regional wall motion abnormality, poor graft flow
 Incomplete revascularization
  Non-graftable vessels
  Known intrinsic disease
 Metabolic
  Hypoxia, hypercarbia ABG, electrolytes, check ventilation
  Hypokalemia, hyperkalemia Electrolytes
 Uncorrected pathology
  Hypertrophic cardiomyopathy TOE Abnormal outflow gradient, SAM
  Valve gradients TOE Abnormal valve gradient
  Shunts TOE Abnormal Doppler jet
 Mechanical issues
  Prosthetic valve function TOE Poor leaflet motion, abnormal gradient
  Intracardiac shunt (ASD, VSD) TOE Abnormal Doppler jet
 Conduction issues
  Bradycardia ECG Heart rate less than 60
  Atrioventricular dissociation ECG Third degree heart block
  Atrial fibrillation ECG, ABG, electrolytes Hypoxia, electrolyte abnormality
  Ventricular arrhythmias ECG, ABG, electrolytes Hypoxia, electrolyte abnormality
  Vasodilation Transpulmonary thermodilation, Swan-Ganz monitoring Decreased systemic vascular resistance
  Hypovolemia Stroke volume monitoring Decreased stroke volume, increased SVV
 Pulmonary hypertension
  Pre-existing elevated pulmonary pressures, hypoxia, hypercarbia, fluid overload ABG Elevated pulmonary artery pressures, hypoxia, hypercarbia, RV distention
 Right ventricular failure
  Elevated pulmonary pressures, inadequate myocardial protection, emboli to native or bypass circulation, fluid overload Swan-Ganz monitoring, ABG, TOE RV distention, poor RV wall motion, elevated pulmonary artery pressure, elevated central venous pressure

ABG = arterial blood gas; ASD, atrial septic defect; ECG, electrocardiogram, RV, right ventricle, SAM, systolic anterior motion of mitral valve leaflet; SVV, stoke volume variation; TOE, transoesophageal echocardiography; VSD, ventricular septal defect. Data taken from [80].