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. Author manuscript; available in PMC: 2010 Aug 4.
Published in final edited form as: Innate Immun. 2009 Aug 26;16(4):226–234. doi: 10.1177/1753425909339237

Fig. 2.

Fig. 2

NF-κB/p65 was activated in GECs when exposed to whole-cell P. gingivalis. In response to P. gingivalis, a time-dependent increase in the NF-κB/p65 activation was demonstrated (15 min, 30 min, 45 min, and 60 min; * to **** P<0.001). Stimulation with IL-1β served as a positive control for NF-κB/p65 activation (P<0.001). Both P. gingivalis pretreated with the protease inhibitor TLCK and blank bacteria medium did not activate NF-κB/p65 in gingival epithelial cells. Triplicate experiments were performed on OKF6/TERT-2 and primary GECs from one donor. *Significant difference (P<0.05). Error bars indicate SD values.