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. Author manuscript; available in PMC: 2010 Aug 4.
Published in final edited form as: Innate Immun. 2009 Aug 26;16(4):226–234. doi: 10.1177/1753425909339237

Fig. 5.

Fig. 5

Analysis of the effect of inhibiting NFAT and NF-κB in P. gingivalis-induced MIP-3α/CCL20 gene expression using real-time PCR. (A) Pre-incubation with the inhibitor for NFAT did not influence P. gingivalis-induced MIP-3α/CCL20 gene expression. (B) In GECs pretreated with the specific inhibitor for NF-κB (MG132), gene expression of MIP-3α/CCL20 was blocked after stimulation with P. gingivalis (*P=0.02). (C) The results showed a dose-dependent decrease of the P. gingivalis-induced MIP-3α/CCL20 gene expression in gingival epithelial cells (5 μM, 10 μM, and 30 μM; *P=0.01, **P<0.001, and ***P=0.006, respectively). Triplicate experiments were performed on OKF6/TERT-2 and primary GECs from two to three different donors. *Significant difference (P<0.05). Error bars indicate SD values.