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. 2010 Jul 15;137(14):2417–2426. doi: 10.1242/dev.047993

Fig. 2.

Fig. 2.

Misexpression of UASkuzDN results in ectopic crossing of apterous neurons. (A-C) Stage 16 apGal4, UASTau-Myc-GFP embryos were stained with a polyclonal antibody against GFP. Anterior is up. In wild-type embryos (A) the ap axons do not cross the midline, whereas misexpression of UASKuzDN driven by apGal4 (B) results in ectopic crossing (arrows with asterisks). (C) Removing one copy of slit in embryos mis-expressing UASKuzDN enhances the crossing phenotype. (D) Quantification shows the percentage of segments in which the ap axons cross the midline. **P<0.0001, *P<0.05 (unpaired Student's t-test). Error bars indicate s.d.