Table I.
Patient | Gender | Age (years) | Antiepileptic medications | Electrode placement | Seizure onset zones on ECoG | Resected areas | Histology |
---|---|---|---|---|---|---|---|
1 | M | 6 | LEV, OXC, ZNS | Rt FPTO | Rt PO | Rt POT | Tumor |
2 | F | 8 | LEV, OXC | Rt FPTO | Not captureda | Rt FP | Gliosis |
3 | M | 8 | OXC | Lt FPTO | Not capturedb | Lt T | Tumor |
4 | F | 10 | OXC, TPM | Lt FPTO | Lt TP | Lt T | Tumor |
5 | M | 10 | LEV, OXC, TPM | Rt FPTO | Rt O | Rt OPT | Dysplasia |
6 | F | 11 | LEV, OXC, VPA | Rt FPTO | Rt FP | Rt FP | Dysplasia |
7 | F | 11 | LEV, OXC | Rt FPTO | Rt T | Rt TF | Dysplasia |
8 | M | 14 | LEV, OXC, TPM | Rt FPTO | Rt PT | Rt PT | Dysplasia |
9 | F | 16 | CBZ | Rt FPTO | Not capturedc | Not applicable | Not Available |
10 | F | 16 | CZP, PHT, TPM | Rt FPTO Lt FP | Rt FPT | Rt FPT | Gliosis |
11 | M | 16 | OXC, TPM | Rt FPTO | Rt T | Rt T | Dysplasia; Hippocampal Sclerosis |
12 | F | 17 | LTG | Lt FPTO | Lt F | Lt F | Gliosis |
13 | F | 17 | LEV, OXC | Rt FPTO | Rt O | Rt O | Gliosis |
14 | M | 17 | OXC | Lt FPTO | Lt T | Lt T | Tumor |
15 | F | 18 | LTG, OXC, ZNS | Rt FPTO | Rt PTO | Rt PTO | Ulegyria |
Frequent interictal spikes were noted in the right inferior frontal‐parietal regions.
Occasional generalized spike‐wave discharges were noted; lesionectomy of the tumor in the left temporal region was performed.
Habitual seizures failed to be captured during chronic ECoG recording; no resective surgery was performed. However, habitual seizures characterized by forced head‐deviation toward the left side were captured during preoperative scalp EEG recording; thereby, delayed ictal discharges were noted over the right hemisphere but further localization of the presumed epileptogenic zone was not tenable in Patient 9.
F, female; M, male; Lt, left; Rt, right; CBZ, carbamazepine; CZP, clonazepam; LEV, levetiracetam; LTG, lamotrigine; OXC, oxcarbazepine; PHT, phenytoin; TPM, topiramate; VPA, valproic acid; ZNS, zonisamide; F, frontal; P, parietal; T, temporal; O, occipital.