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. Author manuscript; available in PMC: 2011 Jul 1.

Published in final edited form as: Cancer Res. 2010 Jun 15;70(13):5567–5576. doi: 10.1158/0008-5472.CAN-09-4510

Fig. 2 — (A) RT-PCR analysis of ESCC and NPC tumorigenic ADAMTS9 stable clone-derived tumors and tumor segregants with eight primer pairs covering the entire ADAMTS9 transcript. Relative localizations of the primer pairs are indicated. ○, positive expression; ●, no expression; , reduced expression. (B) Representative RT-PCR results of the ADAMTS9 transcript regions P4 and P6 in selected tumors derived from the stable ADAMTS9 clones. GADPH served as a loading control. (C) Western blot analysis of the ADAMTS9 knockdown transfectants in two NPC clones and the vector-alone control. (D) Tumor growth kinetics of the two ADAMTS9 knockdown and vector-alone clones and HONE1 cells.

Inline graphic — (A) RT-PCR analysis of ESCC and NPC tumorigenic ADAMTS9 stable clone-derived tumors and tumor segregants with eight primer pairs covering the entire ADAMTS9 transcript. Relative localizations of the primer pairs are indicated. ○, positive expression; ●, no expression; , reduced expression. (B) Representative RT-PCR results of the ADAMTS9 transcript regions P4 and P6 in selected tumors derived from the stable ADAMTS9 clones. GADPH served as a loading control. (C) Western blot analysis of the ADAMTS9 knockdown transfectants in two NPC clones and the vector-alone control. (D) Tumor growth kinetics of the two ADAMTS9 knockdown and vector-alone clones and HONE1 cells.