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. 2010 Apr 9;299(1):L25–L35. doi: 10.1152/ajplung.00228.2009

Fig. 10.

Fig. 10.

Schematic representation of a potential mechanism of IFN-dependent modulation of mitogen-induced protein synthesis in ASM. See discussion for details. mTOR, mammalian target of rapamycin; ISRE, IFN-stimulated response element; IFI16, IFN-γ-inducible protein 16; GAS, IFN-γ-activated sequence.