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LC3-II autophagic forms are produced by Ad-IFNα in cancer cells but not in normal cells which can be inhibited by the autophagy inhibitor, 3-MA. Increases in the LC3-II autophagic forms were seen over time in Ad-IFNα- treated UC-9 cancer cells which could be blocked by exposure of various doses of 3-MA. In contrast no LC3-II was seen in Ad-IFNα treated normal human urothelial cells.
