TABLE 2.
Recovery of known causal QTL GSL genes in GWA mapping
| AGI | Name | 2007 | 2008 |
|---|---|---|---|
| AT1G12140 | GSOX5 | — | — |
| AT1G24100 | UGT74B1 | — | — |
| AT1G62540 | GSOX2 | — | — |
| AT1G62560 | GSOX3 | — | — |
| AT1G62570 | GSOX4 | — | Yes |
| AT1G65860 | GSOX1 | — | — |
| AT2G25450 | GS-OH | — | — |
| AT2G31790 | UGT74C1 | — | — |
| AT4G03050 | AOP3 | Yes | Yes |
| AT4G03060 | AOP2 | Yes | Yes |
| AT5G07690 | MYB29 | — | — |
| AT5G07700 | MYB76 | — | — |
| AT5G23010 | MAM1 | Yes | Yes |
| AT5G57220 | CYP81F2 | — | — |
| AT5G60890 | ATR1/MYB34 | — | — |
| AT5G61420 | MYB28 | — | — |
Shown are genes that have been previously proven to be both genetically polymorphic and control a QTL for GSL accumulation within the 96 accessions used in this analysis (Kliebenstein 2009). Yes, if the gene has ≥2 SNPs showing significant association to one or more GSL traits in the corresponding GWA; —, no significant associations.