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. Author manuscript; available in PMC: 2011 Sep 1.
Published in final edited form as: Toxicol Lett. 2010 May 31;197(3):163–168. doi: 10.1016/j.toxlet.2010.05.017

Fig.2. DEP did not cause direct toxicity to HAEC.

Fig.2

(A) HAEC cells grown to confluence were treated with or without 50ug/ml DEP1 or DEP2 for 4 hours. The supernatant was used to measure the activity LDH as indication of cytotoxicity. DEP did not increase the LDH levels released by HAEC. (B) HAEC cells grown to confluence were treated with or without 50ug/ml DEP1 or DEP2 for 4 hours, HAEC apoptosis was examined by measuring Caspase-3 activity. DEP did not cause significant apoptosis of HAEC after 4 hours of treatment. Camptothecin (CPT) at 10uM was used as positive control of apoptosis. (C= control)