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. Author manuscript; available in PMC: 2011 Aug 1.
Published in final edited form as: Mol Microbiol. 2010 Jun 1;77(3):587–604. doi: 10.1111/j.1365-2958.2010.07234.x

Table 1.

Strains used in this study

Strain Genotype Description and Source
R. oryzae 99–880 Wild-type Clinical isolate (Ibrahim et al., 2007)
R. oryzae M16 pyrF205 Uracil deficient (Skory & Ibrahim, 2007)
R. oryzae PyrF complemented pyrF205::PyrF M16 complemented with a wild-type copy of PyrF at its original locus, this work
R. oryzae GFP1 M16 (pPFtr1-GFP) M16 transformed with a plasmid containing an FTR1 promoter driven GFP (Ibrahim et al., 2007)
R. oryzae FTR1Ko pyrF205, ftr1::PyrF ftr1 knock out, this work
R. oryzae FTR1Inh M16 (pFTRi-pdc intron) FTR1 inhibited by RNAi, this work
R. oryzae Empty M16 (pRNAi-pdc intron) M16 transformed with empty plasmid, this work
HHS Vulnerability Disclosure