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. Author manuscript; available in PMC: 2010 Jul 26.
Published in final edited form as: In Vivo. 2009 Sep–Oct;23(5):653–660.

Figure 3. Analyses of KLF4-related pathways in HNSCC.

Figure 3

A, Example of hematoxylin-eosin-stained section of human oral SCC. B–F, Consecutive sections stained for phosphorylated Akt (B), KLF4 (C), p53 (D), p21 (E) and cyclin D1 (F). Insets in B, C, D, E, F denote the same area of the tumor at higher magnification. Bars =200 μm.