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. 2010 May 18;1(1):e00012-10. doi: 10.1128/mBio.00012-10

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Copyright © 2010 Sánchez-Romero et al.

This is an open-access article distributed under the terms of the Creative Commons Attribution-Noncommercial-Share Alike 3.0 Unported License, which permits unrestricted noncommercial use, distribution, and reproduction in any medium, provided the original author and source are credited.

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FIG 2 — Genome-wide view of SeqA binding at different points in the cell cycle. The figure shows ChIP-chip data for SeqA binding plotted against features of the E. coli genome in the form of a genome atlas. (A) SeqA binding signal generated using unsynchronized cultures of E. coli (time point A); (B) SeqA binding after chromosome replication had been blocked for 1 h (time point B); (C) SeqA binding in cultures where chromosome replication had been reinitiated in synchronicity for a period of 6 min.

FIG 2 — Genome-wide view of SeqA binding at different points in the cell cycle. The figure shows ChIP-chip data for SeqA binding plotted against features of the E. coli genome in the form of a genome atlas. (A) SeqA binding signal generated using unsynchronized cultures of E. coli (time point A); (B) SeqA binding after chromosome replication had been blocked for 1 h (time point B); (C) SeqA binding in cultures where chromosome replication had been reinitiated in synchronicity for a period of 6 min.