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. Author manuscript; available in PMC: 2011 Jul 23.

Published in final edited form as: Circ Res. 2010 May 20;107(2):271–282. doi: 10.1161/CIRCRESAHA.110.219899

Panel A, aortic superoxide levels were reduced in LDLR+/− mice possessing the SM-caPTH1R transgene (n = 4/group). Panel B, pro-MMP9 activity, an index of ROS signaling down-stream of TNF and OPN²³, was reduced in VSMCs from LDLR+/− mice possessing the SM-caPTH1R transgene. Representative zymogram (left) and quantitative digital image analysis (right).

Panel A, aortic superoxide levels were reduced in LDLR+/− mice possessing the SM-caPTH1R transgene (n = 4/group). Panel B, pro-MMP9 activity, an index of ROS signaling down-stream of TNF and OPN²³, was reduced in VSMCs from LDLR+/− mice possessing the SM-caPTH1R transgene. Representative zymogram (left) and quantitative digital image analysis (right).