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. Author manuscript; available in PMC: 2010 Aug 2.
Published in final edited form as: J Pharmacol Exp Ther. 2008 May 2;326(2):369–379. doi: 10.1124/jpet.108.137208

Fig. 6.

Fig. 6

BSO caused oxidative stress by increasing the expression of NADPH oxidase subunits gp91 phox (A) and p47 phox (B) as shown by real-time PCR (n = 7–10) and gp91 phox protein (C) as shown by Western blotting (n = 3–4) in the amygdala. The PDE2 inhibitor Bay 60-7550 (Bay) and the NADPH oxidase inhibitor apocynin reversed the BSO-induced increase in the expression of NADPH oxidase subunits. Values are expressed as means ± S.E.M. #, p < 0.05 compared with control; *, p < 0.05 compared with BSO-treated group.