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. Author manuscript; available in PMC: 2011 Sep 1.
Published in final edited form as: J Pathol. 2010 Sep;222(1):110–116. doi: 10.1002/path.2739

Table 2.

Analysis of SCOUTs for p53 mutations

Sample* Source Exons tested WT/Mut Nucleotide Amino acid Effect Comment
02N Normal E2–E9, E11 WT
02SC SCOUT E2–E11 WT Remote to carcinoma
02Ca CA E2–E4. E6, E8–E11 Mut c.1079G>T G360V missense
04N Normal E2–E11 WT
04SC1 SCOUT E2–E11 WT Remote to carcinoma (Figure 3C)
04SC2 SCOUT E2–E11 WT Remote to carcinoma
04SC3 SCOUT E2–E11 WT Remote to carcinoma
04SC4 SCOUT E2–E11 WT Remote to carcinoma
06N Normal E2–E5, E7, E9–E11 WT
06SC SCOUT E2–E4, E6, E7, E9–E11 WT
10N Normal E2–E11 WT
10SC SCOUT E2–E11 Mut c.659A>C p.Y220S missense Contiguous with carcinoma (Figure 4)
10T STIC E2–E11 Mut c.659A>C p.Y220S missense
10Ca Serous carcinoma E2–E11 Mut c.659A>C p.Y220S missense Contiguous with SCOUT (Figure 4)

Sample numbers are from the original laboratory datasheet. Samples with inadequate DNA amplification were excluded. SCOUT = secretory cell outgrowth; STIC = serous tubal intraepithelial carcinoma; WT = wild type p53 DNA sequence; Mut = p53 mutation detected.