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. 2010 Jul 1;61(13):3589–3598. doi: 10.1093/jxb/erq172

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© 2010 The Author(s).

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/2.5), which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

This paper is available online free of all access charges (see http://jxb.oxfordjournals.org/open_access.html for further details)

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Fig. 4. — The effects of the putative auxin influx inhibitors on subcellular distribution of PIN1:GFP, EYFP:AUX1, and ABCB4:GFP fusion proteins in tobacco BY-2 cells. (A, E, I) non-treated controls. The effects of 1-NOA (20 μM, 3 h) (B), 2-NOA (20 μM, 3 h) (C), and CHPAA (20 μM, 3 h) (D) on the subcellular distribution of PIN1:GFP. The effects of 1-NOA (20 μM, 3 h) (F), 2-NOA (20 μM, 3 h) (G), and CHPAA (20 μM, 3 h) (H) on the subcellular distribution of EYFP:AUX1. The effects of 1-NOA (20 μM, 24 h) (J), 2-NOA (20 μM, 24 h) (K), and CHPAA (20 μM, 24 h) (L) on the subcellular distribution of ABCB4:GFP. Single confocal sections through the cortical cytoplasm (C, Apochromat ×40 1.2 W water immersion objective with a 1 Airy Unit pinhole). Scale bars, 10 μm.