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. Author manuscript; available in PMC: 2010 Aug 16.
Published in final edited form as: J Recept Signal Transduct Res. 2010 Apr;30(2):78–87. doi: 10.3109/10799891003614808

Figure 6.

Figure 6

Translocation of mtCxn-43: (A) immunobloting for Cxn-43 in mitochondrial fractions from WT, WT + Hcy-treated for 6 weeks, NMDA-R1 knockout (NR1-KO)-treated with Hcy, and NR1-KO (control) mice. The COXIV was used as control. (B) Densitometric analysis was performed and plotted as a bar graph. *P < 0.05 compared with WT; #P < 0.05 compared with NR1fl/fl/Cre with Hcy. Data were representative of n = 4 in each group. (C) Degradation of Cxn-43 was measured in myocytes from WT, WT + Hcy-treated for 6 weeks, MMP-9 knockout (MMP-9KO)-treated with Hcy, and MMP-9-KO (control) mice. The β-actin was used as control. (D) The bands were scanned and presented as bar graphs. *P < 0.05 compared with WT; #P < 0.05 compared with MMP-9KO with Hcy. Data were representative of n = 4 in each group.