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. 2010 Aug 23;5(8):e12341. doi: 10.1371/journal.pone.0012341

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This is an open-access article distributed under the terms of the Creative Commons Public Domain declaration which stipulates that, once placed in the public domain, this work may be freely reproduced, distributed, transmitted, modified, built upon, or otherwise used by anyone for any lawful purpose.

This is an open-access article distributed under the terms of the Creative Commons Public Domain declaration, which stipulates that, once placed in the public domain, this work may be freely reproduced, distributed, transmitted, modified, built upon, or otherwise used by anyone for any lawful purpose.

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(A) Mitochondrial ATP production was measured in exponentially growing HCT116 p53^+/+, HCT116 p53^−/−, or stable HCT116 p53^−/− transfectants with p53 wild-type, or S315A/S315D mutants (n = 3; mean ± SEM; **P<0.01). (B) Mitochondrial ATP production was measured in the same set of cells additionally transfected with cyclin B1, Cdk1 or scrambled siRNAs for 24 h (n = 3; mean ± SEM; *P<0.05; **P<0.01). (C) Mitochondrial ATP production was measured in the same set of cells transfected with MTS-EGFP and MTS-RFP, or MTS-cyclin B1 and MTS-Cdk1, or MTS-cyclin B1 and MTS-delCdk1 (n = 3; mean ± SEM; **P<0.01).