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. 2010 Aug 23;5(8):e12341. doi: 10.1371/journal.pone.0012341

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This is an open-access article distributed under the terms of the Creative Commons Public Domain declaration which stipulates that, once placed in the public domain, this work may be freely reproduced, distributed, transmitted, modified, built upon, or otherwise used by anyone for any lawful purpose.

This is an open-access article distributed under the terms of the Creative Commons Public Domain declaration, which stipulates that, once placed in the public domain, this work may be freely reproduced, distributed, transmitted, modified, built upon, or otherwise used by anyone for any lawful purpose.

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DNA-damage irradiation stimulates the gene expression and mitochondrial translocation of p53, cyclin B1 and Cdk1. Mitochondrial cyclin B1 and Cdk1 form a functional complex to phosphorylate p53, which leads to enhanced ATP production and suppression of mitochondria-mediated apoptosis.