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. Author manuscript; available in PMC: 2010 Aug 25.
Published in final edited form as: Hippocampus. 2003;13(3):399–412. doi: 10.1002/hipo.10089

FIGURE 3.

FIGURE 3

Neuronal populations which are typically vulnerable to seizure-induced excitotoxicity are not reduced in rats studied 3 months after developmental febrile seizures (B,D,F) when compared with age-matched controls (A,C,E). A,B: Hilar γ-aminobutyric acid (GABA)ergic interneurons visualized using in situ hybridization (ISH) for GAD67 mRNA. C,D: Hilar mossy cells, demonstrated using immunocytochemistry for AMPA-receptor subunits GluR2/3. E,F: CA1 interneurons, visualized using GAD67-ISH. GCL, granule cell layer; H, hilus; s.o., stratum oriens; s.p., stratum pyramidale; s.r., stratum radiatum. Scale bars = 80 μm in A,B; 50 μ min C–F.