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. Author manuscript; available in PMC: 2010 Sep 8.
Published in final edited form as: Cancer Cell. 2009 Sep 8;16(3):232–245. doi: 10.1016/j.ccr.2009.07.030

Table 2.

Mutation analysis of BCR-ABL1 kinase domain mutations in PAX5-transduced CML cells

Cell Type CD19+ clones AID mRNA [% COX6B] ABL1 exons 4-6 Sequence analysis Clones Amino acid change IC50 [μmol/l Imatinib]
KYO-GFP < 0.01% <0.01 764 GAG→AAG 1/35 E255K 12.1
Wildtype and silent 34/35 0.6
Imatinib-resistant 1/35 (2.9%)

KYO-PAX5/GFP ~0.5% 4.24 ± 0.95 742 CTG→GTG 4/48 L248V 7.0
764 GAG→AAG 5/48 E255K 12.1
944 ACT→ATT 16/48 T315I 16
1078 ATC→GTC 2/48 I360V unknown
1187 CAT→CGT 2/48 H396R 1.8
Wildtype and silent 21/48 0.6
Imatinib-resistant 27/48 (56%)