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. Author manuscript; available in PMC: 2011 Feb 1.
Published in final edited form as: Neuron. 2010 Aug 12;67(3):373–380. doi: 10.1016/j.neuron.2010.07.004

Figure 3. A full-length nompC cDNA rescues the locomotion defects in nompC1/3 mutant larvae and adult flies.

Figure 3

(A) The exon-intron structures of nompC-L and nompC-A aligned with the genomic sequence. Exons are indicated by shaded boxes/triangles and introns by dotted lines.

(B, C) Quantification of larval crawling speed and stride duration with the expression of NompC-L or NompC-L-GFP by different Gal4 drivers. SN, sensory neurons; MD, multi-dendritic neurons; bd/I, bd and class I da neurons.

(D) Quantification of adult fly walking speed with NompC-L expression.

(E) Quantification of stride duration with expression of the truncated NompC, Δ12ANK-GFP and Δ29ANK-GFP. Error bars represent SEM, n≥20. * P<0.01. See also Figure S2, Movie S3 and Table S1.