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. 2010 May 19;104(2):685–694. doi: 10.1152/jn.00228.2010

Fig. 2.

Fig. 2.

A: stimulation of the LPGi evoked a postsynaptic current in CVNs. Application of the N-methyl-d-aspartate (NMDA) and non-NMDA receptor blockers AP-5 and CNQX, respectively, did not alter the evoked inhibitory postsynaptic current (IPSC) as shown in a typical example (A) and in the summary data (B). However, application of strychnine, a glycinergic receptor antagonist, significantly decreased the evoked postsynaptic current (A, typical experiment; B, summary data). Finally, gabazine, a GABAergic receptor antagonist, nearly abolished the activation of this synaptic pathway (A, typical experiment; B, summary data). As shown in C, paired pulse stimulations with a delay of 10 ms evoked postsynaptic GABAergic responses (isolated by focal application of strychnine, AP-5, and CNQX) in CVNs with a constant latency. +P < 0.05. In this and all subsequent figures, arrow indicates electrical stimulation (unless other stated), ■ with asterisks indicates statistically significant differences, *P < 0.05, **P < 0.01, and ***P < 0.001.