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. Author manuscript; available in PMC: 2010 Dec 1.
Published in final edited form as: Nature. 2010 Jun 10;465(7299):793–797. doi: 10.1038/nature09135

Figure 2.

Figure 2

The HSC response to M. avium infection is dependent upon intact IFNγ signaling.

(A) IFNγ levels in bone marrow supernatant were quantified by cytokine bead array at baseline and 4 weeks postinfection with M. avium. n=3-6. (B) BrdU incorporation by HSCs (KSL CD150+) of naïve and M. avium-infected WT, Ifngr1-deficient, Stat1-deficient, and Ifnar1-deficient mice was quantified. n=3-6. (C) Absolute number of HSCs (KSL CD150+) in the whole bone marrow of naïve and infected WT, Ifngr1-deficient, Stat1-deficient, and Ifnar1-deficient mice was determined. n=3-7.