. Author manuscript; available in PMC: 2011 Oct 1.

Published in final edited form as: Dev Biol. 2010 Jul 14;346(1):25–38. doi: 10.1016/j.ydbio.2010.07.008

Table 2.

Disruption of Pofut2 gene resulted in embryonic lethality by E 10.5

Cross	Stage examined	genotype			Res sites	Litter total	% Hm (p)
Cross	Stage examined	Wt	Het	Hm	Res sites	Litter total	% Hm (p)
RST434 × RST434^*	P 15	35	60	0		95	0
RST434 × RST434^**	E 10.5	18	43	0	19	80	23.8 (0.79)
RST434 × RST434^#	E 7.5	18	27	15	0	60	25 (1.00)
RST434 × RST434^#	E 6.5	11	27	13	0	51	25.5 (0.94)
RST434 × 699Lex^$	E 7.5	ND	ND	14	0	52	26.9 (0.75)
699Lex × 699Lex^$	E 7.5	ND	ND	29	0	114	25.4 (0.91)

Genotype determined by PCR (Materials and Methods)

^**

Resorption sites (Res sites) were counted and surviving embryos genotyped by PCR.

Genotypes were inferred from the intensity of X-gal staining and embryo morphology. The relationship between these characteristics and genotype was verified by PCR (Materials and Methods).

homozygous genotype inferred from mutant morphology. Wt, wild type; Het, heterozygous; Hm, homozygous; nd, not determined.