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Site of action of glycoprotein IIb/IIa inhibitors.
Aggregation requires activated platelets, glycoprotein receptor IIb/IIIa (GP), and fibrinogen. The latter acts as a bridge that attaches platelet to each other forming the initial hemostatic plug. The current ACC/AHA guidelines recommend the use of GP IIb/IIa antagonists in high-risk patients with STEMI-ACS and planned percutaneous coronary intervention.
