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The ctn-1 encodes a homologue of mammalian α-catulin. Shown is the alignment between predicted amino-acid sequences of CTN-1 and human α-catulin (41% identity). The molecular lesions of five ctn-1 alleles were determined. Four alleles have mutations leading to premature stop codon (marked with ‘*') and one allele (marked with an arrow) disrupts a splice donor site leading to a frame shift after amino-acid Y92 and then a stop codon (CFNGQPIMCM STOP).
