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Lesioning the Schaffer collaterals prevented the large amplitude AP-dependent release in CA1 area during H/H. (A) sEPSCs recorded from CA1 in a CA3/CA1 pathway lesioned brain slice before, during transient H/H, and after recovery. (B) Frequency of mEPSC increases significantly during H/H, but the amplitude of mEPSCs did not change during transient H/H (n=6).
