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. Author manuscript; available in PMC: 2011 Oct 1.
Published in final edited form as: Mol Oral Microbiol. 2010 Oct;25(5):305–316. doi: 10.1111/j.2041-1014.2010.00582.x

Figure 2.

Figure 2

Porphyromonas gingivalis induces human platelet aggregation. (A) P. gingivalis 381 (P.g) or DPG3 (DPG3) at 108 colony-forming units (CFU)/ml, was added to a suspension of human platelets (2 × 108/ml) and stirred continuously for 10 min in a PAP-4 aggregometer. The % aggregation relative to a collagen control is indicated. *P < 0.001 vs. collagen control. (B) P. gingivalis 381 (P.g) or DPG3 (DPG3) (108 CFU/ml) was added to suspensions of human whole blood (final platelet concentration, 2 × 108/ml) and stirred continuously in a whole blood aggregometer and formation of platelet neutrophil aggregates was determined by flow cytometry. Black = no P. gingivalis addition; gray = + P. gingivalis. *P < 0.001 vs. unchallenged.