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. Author manuscript; available in PMC: 2010 Oct 30.
Published in final edited form as: Mol Cell Endocrinol. 2009 Jul 15;310(1-2):11–20. doi: 10.1016/j.mce.2009.07.001

Table 1. Skeletal parameters altered by disruption of FSH in the HPG axis.

Genotype Reproductive effects Skeletal effects Following OVX Reference

F ORKO Immature ovary development, thread-like uteri Spinal kyphosis, vertebral compression (4 mo) Decreased BMD, BV/TV (spine, femur) 2, 3, 4
Decreased circulating estrogen Decreased BMD, Tb BV, ObS/BS, MAR, BFR at 3-4 Increased OcS/BS
Increased FSH, LH mo. (spine, femur) Decreased testosterone
10-fold increase in testosterone at 3-4 mo. Decreased ex vivo osteoclastogenesis OVX effect is mimicked by androgen receptor blockade or aromatase inhibition
Inhibin – not determined

FSHβ +/- Normal ovaries and uteri Decreased osteoclast number Not determined 1, 3
Normal estrogen Decreased bone resorption
50% decreased FSH Increased Tb BV, Tb N (spine, femur)
Inhibin, testosterone, LH – not determined No changes in serum osteocalcin, MAR

FSHβ -/- Atrophic ovaries, Thread-like uteri No bone loss Not determined 1, 3
Decreased estrogen Increased BMD (femur)
Decreased FSH; Increased LH Increased BV/TV, Tb N
Inhibin, testosterone – not determined

References: 1. Kumar, 1997; 2. Danilovich, 2000, 2002; 3. Sun, 2006; 4. Gao, 2007

BMD, bone mineral density; Tb BV, trabecular bone volume; ObS/BS, osteoblast Surface/bone surface; MAR, mineral apposition rate; BFR, bone formation rate; BV/TV, bone volume/total volume; OcS/BS, osteoclast surface/bone surface; Tb N, trabecular number