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. 2010 Sep 10;192(21):5767–5777. doi: 10.1128/JB.00624-10

FIG. 4.

FIG. 4.

FliZ controls the magnitude of SPI1 gene expression and the dynamics of type 1 fimbrial gene expression. (A to C) Deleting FliZ represses flagellar and SPI1 gene expression and accelerates the induction of type 1 fimbrial genes. PflgA (A), PhilA (B), and PfimA (C) promoter activities in the wild type and the ΔfliZ mutant are shown. (D to F) Overexpressing FliZ increases the magnitude of flagellar and SPI1 gene expression and delays the induction of type 1 fimbrial genes. PflgA (D), PhilA (E), and PfimA (F) promoter activities in the wild type and the ΔfliZ mutant constitutively expressing FliZ from a PLtetO-1 promoter on a plasmid (pFliZ) are shown. Experiments were performed as described for Fig. 2.