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. 2010 Sep 10;192(21):5767–5777. doi: 10.1128/JB.00624-10

FIG. 6.

FIG. 6.

RtsB controls the dynamics of flagellar and type 1 fimbrial gene expression. (A to C) Deleting RtsB increases the duration of flagellar gene expression and slows the induction of type 1 fimbrial genes. PflgA (A), PhilA (B), and PfimA (C) promoter activities in the wild type and the ΔrtsB mutant are shown. (D to F) Overexpressing RtsB inactivates flagellar gene expression and accelerates the induction of type 1 fimbrial genes. PflgA (D), PhilA (E), and PfimA (F) promoter activities in the wild type and the ΔrtsB mutant constitutively expressing RtsB from the constitutive PLtetO-1 promoter on a plasmid (pRtsB) are shown. Dynamic luminescence experiments were performed as described for Fig. 2. (G) RtsB regulates type 1 fimbrial gene expression through FliZ. PfimA promoter activities in the wild type, the ΔrtsB mutant, the ΔfliZ mutant, the ΔfliZ ΔrtsB mutant, and the ΔrtsB and ΔfliZ ΔrtsB mutants constitutively expressing RtsB are shown. Endpoint fluorescence experiments were performed as described for Fig. 5.