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Cardiomyocyte expression of SUR2A in MLC2A mice results in smaller infarct sizes. Isolated hearts from SUR2 null (n = 13) and MLC2A (n = 7) mice were exposed to 40 min of “no-flow” ischemia via a standard Langendorff setup. After 60 min of reperfusion, hearts were sectioned and stained with 1% 2,3,5-triphenyltetrazolium chloride (TTC) to visualize infarct size. Restoration of cardiomyocyte SUR2A expression resulted in increased resistance to ischemia [area at risk (AAR)]. *P < 0.05.
