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. 2010 Aug 18;104(4):1841–1848. doi: 10.1152/jn.00442.2010

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Fig. 1. — Application of the acetylcholinesterase inhibitor neostigmine (10 μm) significantly decreased the peak amplitude of the excitatory synaptic response to cardiac vagal neurons (CVNs). A typical experiment is shown in the top traces (A), with a ▴ indicating peak synaptic event and the summary data from 8 experiments is shown in B. C: scatterplot of percentage maximum evoked excitatory postsynaptic current (eEPSC) amplitude from 8 experiments. Neostigmine (10 μm) decreased the glutamatergic neurotransmission to cardiac vagal evoked by trigeminal afferent stimulation from −38.1 ± 3.9 to −15.8 ± 2.3 pA (by 58.5 ± 3.7%, n = 8, P < 0.05).