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. Author manuscript; available in PMC: 2010 Oct 21.
Published in final edited form as: Epileptic Disord. 2010 Mar 1;12(1):76–80. doi: 10.1684/epd.2010.0294

Figure 1.

Figure 1

Polysomnogram shows stridor in the sonogram (channel 11) during 5-minute (A) and two consecutive 60-second epochs (B, C). During N2 sleep, recurrent periods of stridor were consistently demonstrated as deflections in the “Snore” Channel 11. Mild transient oronasal airflow limitation and altered respiratory effort occurred in relationship to the audible stridor without associated arousal or oxyhaemoglobin desaturation (A, B). A spontaneous arousal is seen in the latter one-third section of A, but no clearly corresponding respiratory effort-related arousals (RERAs) were seen during or following periods of stridor, and no oxyhaemoglobin desaturations occurred. Arousal index was within normal limits at 5.6 per hour, suggesting that upper airway resistance syndrome was unlikely. Sensitivity 15 uV/mm, TC 0.3 sec, HFF 35 hertz.