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. Author manuscript; available in PMC: 2011 Feb 1.
Published in final edited form as: Dev Dyn. 2010 Feb;239(2):548–558. doi: 10.1002/dvdy.22201

Figure 4. Somite malformation in ChCh and Sip1a compromised embryos can be rescued by reduction of FGF8.

Figure 4

(A) Dorsal views of 10 somite stage and (B) 12 somite stage living embryos, anterior to the top. Somites are narrower at A/P axis, broader at mediolateral axis in wild type and ace heterozygous chch morphants, but not ace homozygous chch morphants (A). ace homozygous sip1a ATG morphants does not have somite phenotype but wild type and ace heterozygous sip1a morphants still have the somite defects. (B). Horizontal and vertical red dotted line spans the first five somites of the control injected embryo width and length for comparison to the first five somite of morphant embryos which is indicated with a black line. Embryos were genotyped for the ace allele after imaging.