Table 6.
Effect | Estimate | SE | LPR | % |
---|---|---|---|---|
aA | −0.013 | 0.027 | 0.20 | – |
dA | 0.046 | 0.034 | 0.76 | – |
iA | −0.009 | 0.027 | 0.13 | – |
aB | −0.006 | 0.029 | 0.08 | – |
dB | −0.002 | 0.036 | 0.02 | – |
iB | 0.017 | 0.029 | 0.25 | – |
aa | −0.097 | 0.034 | 2.35 | 0.50 |
ad | −0.040 | 0.047 | 0.41 | – |
ai | −0.022 | 0.035 | 0.27 | – |
da | 0.101 | 0.047 | 1.52 | 0.27 |
dd | 0.039 | 0.064 | 0.27 | – |
di | 0.153 | 0.046 | 3.10 | 0.62 |
ia | −0.021 | 0.036 | 0.26 | – |
id | −0.028 | 0.048 | 0.25 | – |
ii | 0.148 | 0.034 | 4.81 | 1.16 |
The 15 genetic-effect estimates were fitted using the regression model shown in Eq. 12 and the genotypic index scores shown in Table 1. The single-locus effects with the subscript A correspond to the effects of epiEG3.1 while those with the subscript B correspond to the locus epiEG6.1. Together, the significance test of the epistatic interaction has an LPR of 5.54. The combined effects of the locus explain 2.8% of the phenotypic variance (2.5% for significant effects). An LPR value of 1.3 corresponds to a p value of 0.05. Significant effects are highlighted in boldface. Other details are given in Table 5