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. Author manuscript; available in PMC: 2011 Dec 15.
Published in final edited form as: Dev Biol. 2010 Oct 16;348(2):210–221. doi: 10.1016/j.ydbio.2010.10.003

Table 2.

Suppression of the precocious alae phenotype of lin-14 and lin-28 mutants by rbm-28 (RNAi). The indicated worm strains were cultured at 25°C on vector control (white rows) or rbm-28 (RNAi) (gray rows) and scored for alae formation (incomplete refers to non-contiguous alae ridges due to variable seam cell differentiation) at the L4 stage based on gonad development.

Strain scored at L4 stage based on size and gonad development No alae Incomplete alae Full alae
N2 (n=17) 100% 0% 0%
N2; rbm-28 RNAi (n=11) 100% 0% 0%
lin-14(n179) (n=13) 15% 54% 31%
lin-14(n179); rbm-28 RNAi (n=13) 86% 7% 7%
lin-14(n179);lin-4(e912) (n=16) 100% 0% 0%
lin-14(n179);lin-4(e912); rbm-28 RNAi (n=10) 90% 10% 0%
lin-4(e912) (n=15) 100% 0% 0%
lin-4(e912); rbm-28 RNAi (n=8) 100% 0% 0%
lin-28(n719) (n=18) 28% 22% 50%
lin-28(n719); rbm-28 RNAi (n=12) 100% 0% 0%