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. Author manuscript; available in PMC: 2011 Apr 1.
Published in final edited form as: Circ Res. 2010 Aug 12;107(8):1021–1031. doi: 10.1161/CIRCRESAHA.110.218966

Figure 5. ATM+/− mice show a defect in oxidative phosphorylation on metabolomic screening.

Figure 5

A, High-resolution 500 MHz 1H-NMR spectra of liver extracts from ATM+/+/ApoE−/− and ATM+/−/ApoE−/− mice. Top, β-Hydroxybutyrate, lactate, and alanine peaks are labeled. Bottom, Multiple peaks show the glucose region in NMR spectra. B, β-Hydroxybutyrate concentrations in liver, pancreas, and plasma of ATM+/−/ApoE−/− mice. Data are shown as relative changes in ATM+/+/ApoE−/− compared with ATM+/+/ApoE−/− mice (means±SEM). *P<0.05, **P<0.01. C, Major lipid changes in the liver and pancreas. Data are shown as relative changes in ATM+/+/ApoE−/− compared with ATM+/+/ApoE−/− mice (means±SEM). *P<0.05, **P<0.01, ***P<0.001. Black bars indicate ATM+/+/ApoE−/−; unfilled bars, ATM+/−/ApoE−/−.